's general knowledge of pathogenesis of bacterial pneumonia

's general knowledge of pathogenesis of bacterial pneumonia
The immunity if excretes the function of the system, body fluid and cell immunological function in sucking filtration and wet melting, epiglottis and cough reflex, bronchus cilium mucus of the gas while defending the mechanism, make the trachea, bronchus and alveolus tissue keep the aseptic state.
The immunological function is damaged ' Such as catching cold, hunger, tired, drunk, go into a coma, poison gas suck and the intersection of hypoxemia and disease, lung edema, uremia, malnutritive, virus infect and employ glucocorticoid, artificial the intersection of air and dish, nasogastric tube,etc.) Enter lower the intersection of pathogen and the intersection of virulence and relatively better or quantity of respiratory tract relatively a long time, pneumonia incident.
The bacterium invades the way to mainly suck (aspiration) for the field planting fungus of mouth swallowing department Suck (inhalation) with the disease-carrying aerosol ,The former is the most important morbidity mechanism of pneumonia, especially the pneumonia and blue negative bacillus pneumonia of leather in the hospital. The bacterium plants, is infected with and spread in the neighbouring position directly or the other positions sow the scattered ones rarely by the blood one.
The typical pathologic change of pneumonia and streptococcus pneumonia is divided into 4 issues: It is early to mainly appear for hydropic liquid and slurry; Ooze out for the erythrocyte in middle period; Have a large number of leucocyte and phagocyte, collect, accumulate later stage, lung organize sudden change; Absorb dissipating for pneumonia finally. After the antibacterial medicine is employed, it is already rare to develop to big whole leaf inflammation, typical lung consolidation is less, and instead of a section of sex inflammation of the lung.
There is no destruction or necrosis of alveolus wall and other lung structure in whole pathological change in the pathologic characteristic, the lung is organized to totally return to normal without leaving over fiber or pulmonary emphysema after pneumonia is dissipated. Although other bacterial pneumonia have the above-mentioned similar pathologic course, the great majority are destroyed with the alveolus wall at various degrees.
Gold the intersection of Portugal and fungus, in the pneumonia, whom bacterium produce solidify alcohol, can form, protect membrane in order to resist phagocyte, kill function outside the body in fungus, and can cause what the lung is organized necrosis in release of different enzymes and abscess forming. Pathological change is invaded and or can form empyema or suppuration pneumothorax to pierce through the pleura. Pathological change can form the lung gasbag while dissipating.
Blue negative bacillus pneumonia of leather is mostly the small leaf pneumonia of dual sides, often occur frequently in the necrosis nature cavity or abscess, empyema can take place to some patients. It is often incomplete to dissipate, can cause fibrous hyperplasia, residual purulent cooking stove and bronchiectasis.
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