The leucocyte that the bacterium secretes lies between 10 plain _'s general knowledge of big enteritis that can prevent and cure the little mouse
The scientists of Belgium and Germany use the lactic acid milk coccus, there is a cell factor leucocyte against inflammation function that will lie between 10 plain inflammatory positions that delivers to the little mouse of big enteritis directly. The lactic acid milk coccus is a kind of harmless fungus which uses the fermented food. (Science 2000; 289:1311-1312,1352-1355)
The scientists of Belgium and Germany use the lactic acid milk coccus, there is a cell factor leucocyte against inflammation function that will lie between 10 plain inflammatory positions that delivers to the little mouse of big enteritis directly. The lactic acid milk coccus is a kind of harmless fungus which uses the fermented food.
Dr. Steidler and his colleague of Belgian Ghent university published the scientific magazine on August 25 Report not this China last mankind kinds of technology,then last a prices getting better high-efficient it than, therapeutic method that be employed for a long time.
Steidler research group that Dr. lead utilize genetic engineering make the intersection of lactic acid and the intersection of milk and coccus can secrete leucocyte, lie between plain 10. They inject get experimental big the intersection of enteritis and the intersection of model and little stomach of mouse bacterium of transformation this kind, found finally the severity of the little mouse pathological change dropped by 50%. It is plain lie between by mice little 10 by leucocyte usually will no enteritises big will be taken place,and it is if you can't secrete by leucocyte, lie between to can be prevented these mice littles from lasting enteritises big a plain lactic acid milk coccus 10 not to inject.
In addition, Dr. Steidler and his colleague also report, the dosage that they use is only to obtain the necessary leucocyte of systemic therapeutic effect and lie between a part of plain dosage 10.
Dr. Shanahan of the Irish Cork university hospital points out in the comment written for this article, because systemic side effect and lacking the organ specificity, some other kinds of therapeutic tactics of the inflammation enteric disease have been employed the restriction by the very great one. Though security and validity of such new method which Dr. Steidler and his colleague design await further proving conclusively, but it has undoubtedly opened up a brand-new route to carry on therapeutic operation to the intestinal bacterium's composition.
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