The senilis of cell can bring out atherosclerosis. 's general knowledge

The senilis of cell can bring out atherosclerosis. 's general knowledge
(Reuter's medical news) One research that issue at latest one the intersection of lancet and magazine show, atherosclerosis might premature old and feeble and relevant with cell.
Leichester university Nilesh J of Britain. Dr. Samani and his colleague point out, the telomere is the bit of DNA of the terminal chromosome, usually increase and shorten with age. And discover newly, the telomere of the disease patient of coronary artery is shorter than that of normal people.
Researchers have carried on comparison to the telomeres of 20 normal person and 10 serious atherosclerosis patient's leucocytes, find the intersection of telomere and enzyme of the latter nearly than normal old and feeble 9 year old too early finally. They infer their reasons may lie in: Coronary artery disease dangerous the factor ' Such as diabetes or hypertension) Have caused telomeres to shorten. But if is really so, it is not significant for the telomere to shorten in the disease process of atherosclerosis, it is only only that one is continued and discovered alikely.
Another kind of possible reason is, the telomere is shortened and made of inherent cause, or come from the excessive replacement of life early stage cells, have already existed before atherosclerosis happened. Two evidences support this view at present: One is that some coronary artery disease patients have hereditary inclination different to explain; Low weight born children, grow, can run into normal child in the intersection of children and issue secondly ' Mean cells are upgraded and accelerated) ,It is extremely apt to suffer from coronary artery disease after its adult.
Seeing that the telomere has important influence on the cell function, Dr. Samani hopes this research can be in order to expound coronary artery disease has illness coming on the mechanism offers the basis.
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